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Sökning: LAR1:lu > (1980-1994)

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1.
  • 10-tals student
  • 1994
  • Samlingsverk (redaktörskap) (populärvet., debatt m.m.)abstract
    • Återutgivning av redaktören och studentarkivarien Ture Sjögrens artikelserie "10-tals student", ursprungligen publicerad i Lunds Veckoblad 1961.
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  • Abdul-Rahman, A, et al. (författare)
  • Local cerebral blood flow in the rat during severe hypoglycemia, and in the recovery period following glucose injection
  • 1980
  • Ingår i: Acta Physiologica Scandinavica. - Wiley-Blackwell. - 0001-6772. ; 109:3, s. 307-314
  • Tidskriftsartikel (refereegranskat)abstract
    • In order to assess the influence of severe hypoglycemia on local cerebral blood flow (1-CBF) artificially ventilated rats, maintained on 70% N2O, were injected with insulin to provide either an EEG pattern of slow-wave polyspikes, or cessation of spontaneous EEG activity for 5, 15 or 30 min ("coma"). In other animals, glucose was injected at the end of a 30 min period of "coma" and 1-CBF was measured after recovery periods of 5, 30, 90, or 180 min. Local CBF was measured autoradiographically with 14C-iodoantipyrine as the diffusible tracer. In the slow-wave polyspike period 1-CBF was increased in most of the structures studied, and reached values that were 1.4 to 3.2 times greater than control. In many structures, cessation of EEG activity was accompanied by a further increase in 1-CBF, with some structures (thalamus, hypothalamus, pontine gray, and cerebellar cortex) showing flow rates of 400--500% of control. The increase in 1-CBF was unrelated to arterial hypertension, hypercapnia, or hypoxia. 5 min after glucose injection the hyperemia persisted in only some of the structures studied; in others, the 1-CBF were close to, or below, control values. During the subsequent recovery period 1-CBF was markedly reduced with some structures (cerebral cortical areas, hippocampus, and caudate-putamen) showing flow rates of only 20--35% of control. In others, notably pontine gray and cerebellar cortex, secondary hypoperfusion was never observed. The hypoperfusion was unrelated to arterial hypertension, hypocapnia, or increase in intracranial pressure. It is concluded that, like hypoxia and ischemia, substrate deficiency due to hypoglycemia is accompanied by vasodilatation in the brain. Furthermore, like long-lasting ischemia, severe hypoglycemia is followed by a delayed hypoperfusion syndrome that, by restricting oxygen supply, may well contribute to the final cell damage incurred.
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  • Aberg, T, et al. (författare)
  • Cerebral function monitoring in rats with a critical hepatic injury treated with pneumatic antishock garment and infusion
  • 1989
  • Ingår i: Journal of Trauma. - Lippincott Williams and Wilkins. - 0022-5282. ; 29:2, s. 168-174
  • Tidskriftsartikel (refereegranskat)abstract
    • Twenty-nine rats were subjected to a severe standardized hepatic injury and divided into four groups. In addition to controls, the animals were treated with PASG inflated to 40 mm Hg, PASG and infusion of Ringer's acetate, or PASG and infusion of Ringer's acetate and Dextran 70 in combination. The aim of the infusion therapy was to stabilize the mean aortic blood pressure at 60 mm Hg. PASG significantly prolonged the survival time and the time during which a sensory evoked response could be observed. The PASG also prolonged the time before the EEG amplitude began to decrease or a burst-suppression pattern appeared in the EEG. Intravenous infusion of Ringer's acetate did not prolong these times compared to when PASG was used alone; when Dextran 70 was added to the infusion therapy these times were reduced. Changes in the EEG were recorded at a mean aortic pressure of 60 mm Hg when infusions were given, whereas the aortic pressure had to fall to 40 mm Hg before any changes could be observed when no infusions were used.
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  • Abrahamson, Magnus (författare)
  • Cystatins
  • 1994
  • Ingår i: Methods in Enzymology. - Academic Press. - 0076-6879. ; 244, s. 685-700
  • Tidskriftsartikel (refereegranskat)
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